ME3738 is a new compound that attenuates liver disease in several models of acute and chronicliverinflammation.
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Background: Hepatitis C infection induces an acute and chronicliverinflammation that may lead to cirrhosis, liver failure, or hepatocarcinoma.
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One patient with recurrenthepatitis B developed chronic severe hepatitis B despite treatment.
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The severity of recurrenthepatitis C virus (HCV) is likely related to several factors.
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Methods: Twenty-nine cases of recurrenthepatitis C and 26 cases of ACR were included in this study.
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One patient died of recurrenthepatitis C and graft failure without evidence of tuberculous infection at death.
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Conclusions: Improving the outcome of recurrenthepatitis C may be achieved by reducing overall immunosuppression and avoiding abrupt variations in immunosuppression.
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Gilead said chronichepatitis B affects more than 400 million people worldwide.
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Methods: One hundred forty-two unselected patients with chronichepatitis C were included.
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Conclusions: Entecavir therapy showed excellent efficacy in nucleoside-naive chronichepatitis B patients.
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Background and aim: Entecavir has demonstrated clinical efficacy for chronichepatitis B.
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This review focuses on the current treatment concepts for chronichepatitis B.
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Hepatic form is manifested by acute or chronichepatitis, steatosis or cirrhosis.
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Crescentic glomerulonephritis was diagnosed in two chronichepatitis B surface antigen carriers.
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All biopsies showed chronichepatitis, of which 2 were associated with cirrhosis.
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Methods: One hundred and twelve patients with biopsy-proven chronichepatitis C were enrolled.
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Aim: Predict the elimination of chronichepatitis C in Catalan prisons.
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The course of chronichepatitis C is variable, but usually fibrosis advances slowly.
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Comprehensive management of chronichepatitis C takes into account cofactors of disease progression.
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Twelve patients with chronichepatitis C were included in the study.
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Despite the declining incidence of acute HCV infection, chronichepatitis C is common.
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Current therapy for HCV-related chronichepatitis is based on the use of interferon.
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A hallmark of chronichepatitis B is a dysfunctional HBV-specific T cell response.